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359 Owing to the difficulty in accessing certain inflamed tissues, our understanding of the abundance, phenotype, and functional status of Tregs across different tissues remains limited, and the impact of distinct inflammatory microenvironments on Tregs also remains unclear

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Furthermore, recent studies identify the involvement of a kidneybrain axis, wherein renal dysfunction may exacerbate neurodegeneration through impaired clearance of neurotoxic metabolites, chronic systemic inflammation, and metabolic dysregulation [32, 33, 34]

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