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doi: 10.1016/j.biopha.2024.116697 151 LiuYXuTMaZZhangCXuMLiQet al

siGAPDH) = 0.9552)

This article systematically reviews the evidence for a role of glutathione redox in the aetiology of autism spectrum disorders and considers the research questions: 1) Is there an association between metabolites/co-factors/genes/enzymes of the -glutamyl cycle or trans-sulphuration pathway and autism spectrum disorders

Concurrently, NF-B-driven inflammatory signaling was inhibited by TNF- expression regulation, which reduced cytokine-mediated oxidative stress and hepatocellular damage