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e-SPEN Eur e-J Clin Nutr Metab 2(6):e116e119 Kamat P, Vacek J, Kalani A, Tyagi N (2015) Homocysteine induced cerebrovascular dysfunction: a link to Alzheimers disease etiology

116 GruhlkeM

Secondary endpoints included LFC responders ( 5% absolute LFC reduction, 30% relative LFC reduction), the changes in noninvasive imaging and serum markers of liver injury and fibrosis, and the changes in lipids