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While GSH has long been recognized as a key brain antioxidant, its role in neurodegenerative diseases is now being unraveled with mechanistic clarity: future research will likely focus on activating astrocytic Nrf2 signaling to enhance glutamate cysteine ligase (GCL)-mediated GSH synthesis, exploring the role of MCT1/2 transporters in neuronal GSH precursor supply, and dissecting how oxidative stress modulates microglial activation via pathways like NF-B-dependent proinflammatory cytokine secretion (Sun et al., 2022)

Furthermore, its potential in treating various diseases, particularly neurodegenerative and respiratory diseases, requires further investigation

These processes express the P2RY12 receptor and make direct contact with smooth muscle cells, pericytes, and endothelial cells of the vasculature (Csszr et al., 2022)

The present study aims at identifying key metabolites and metabolic pathways of IHD that may allow for an earlier intervention and/or a more effective approach to treatment