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This also reveals a critical interdependence between vaginal bacterial translocation, uterine microecology, and endometrial health

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It works by disrupting the interaction between the FOXO4 protein and p53, a key regulator of cell survival

An additional uncertainty is the specicity of the modication, that is, whether the modi- ed cysteine residue is glutathionylated in vivo, rather than nitrosylated or a target of TRX (or some combina- tion of these modications)