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Thus, truncation of the C-terminal L585 residue of the GLP-1 fusion containing WT albumin reduces its cellular recycling in vitro and plasma half-life in vivo, which may be compensated for by QMP engineering of albumin, irrespective of the presence of L585

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Cystine deprivation does not promote mitochondrial oxidative stress To confirm that the persistence of respiratory function in NSCLC was not associated with mitochondrial stress as has been previously reported in other contexts 3,15,22,23 , we interrogated the mitochondrial redox state in response to cystine starvation

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