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Koike, Y., Yozaki, M., Utani, A

[54] was cited in PubMed-indexed journals only 3 times prior to 2006 [55-57], all within the context of understanding the physiology of ASD, not the cause

Isatuximab interacts with a discontinuous epitope partially overlapping the catalytic site, whereas Daratumumab binds to two continuous amino acid sequences situated outside the enzymatic active site of CD38 ( i.e