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Genetic predisposition, co-existing mental health disorders, prior substance abuse, and environmental influences such as peer pressure or high academic and professional demands are all potential contributors

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As there has recently been increasing interest in inflammatory COPD phenotypes, namely eosinophilic versus neutrophilic COPD, we want to note that eosinophils were not detected in our preclinical smoke model which is in line with previous findings ( Overall, we have demonstrated that smoking provokes an excessive recruitment and accumulation of pro-inflammatory and tissue remodeling associated MoAM in the airspace

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