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Furthermore, the level of GLU in the hippocampus was significantly decreased ( F (5, 24) = 6.056, p = 0.001, W = 2.129, p = 0.097) in the F2 group compared to the control group

These findings offer promising insights into developing new treatments for BL by targeting the ATF4-CHOP-CHAC1 pathway (Wang et al., 2019)

Although pathogenic variants of FH have been identified and provide precise genetic and surveillance programs to HLRCC families, there is no association between mutation sites and clinical outcomes in patients [17,18,19]