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Indeed, in vitro studies showed that cells expressing the sst 5 TMD2 mainly respond to cortistatin, whereas those expressing sst 5 TMD4 were exclusively activated by somatostatin and those bearing the sst 5 TMD1 responded to both ligands (Cordoba-Chacon et al., 5 TMD4, while somatostatin activates the sst 5 TMD5 (Duran-Prado et al., Somatostatin in the brain exerts a wide variety of physiological functions besides the initially described inhibitory effect on growth hormone release

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The reason for these apparently anomalous, possibly counterintuitive results [10] is worth highlighting because they can be diagnostically useful

While aflatoxins primarily cause immune suppression, the resulting immune imbalance can sometimes trigger autoimmune responses, where the immune system attacks the body's own tissues (Kraft et al