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These single-cell expression profiles systematically delineate the spatiotemporal regulation patterns of ferroptosis-suppressing signature genes within the renal disease microenvironment, providing direct cellular and molecular evidence for elucidating the mechanisms of ferroptosis imbalance in AKD and CKD pathogenesis, while offering important references for subsequent research on intervention strategies targeting ferroptosis-suppressing pathways in kidney diseases

Supplementary Material Acknowledgement This project was supported by Baylor University and the University of Texas at San Antonio

Some individuals report that fat loss becomes more apparent in specific regions, particularly abdominal adipose tissue, consistent with preclinical data showing preferential visceral fat reduction

This observation suggests the critical role of GPX3, a vital selenoprotein, in safeguarding proximal tubular epithelial cell membranes against oxidative damage during renal IR injury [51]