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2.4.4 Tumor: endothelial cell scavenger receptors and tissue crosstalk In SLE, the endothelium has been shown to be in a dysregulated state, even during low disease activity (246)

As such, our work suggests a novel mechanism, regulated by C32E8.9 , is required for the beneficial longevity effects of mitochondrial translation inhibition, without altering UPR mt