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Future multicenter studies with larger cohorts and longitudinal follow-up are warranted to confirm these findings

Relative abundance of SLC25A39 and GCLM transcripts in HEK-293T cells treated with BSO (1 mM) for the indicated days using quantitative reverse transcription PCR (RT-qPCR), normalized to transcripts of B-ACTIN

128 Moreover, the decreased lactylation level of HDAC1K412 promotes FSP1 mRNA degradation, increasing the susceptibility of colorectal cancer to ferroptosis

Impaired homocysteine metabolism-mediated moderate hyperhomocysteinemia (caused by folate deficiency, vitamin B 6 deficiency, or by methionine excess) might thus have primarily pro-oxidant effects resulting from an altered extra- and intracellular redox potential that can affect essential vascular functions