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Moreover, preclinical RF models exhibit significant limitations: UUO and I/R models induce acute severe fibrosis that does not mimic the gradual progression of human CKD-related RF and lacks comorbidities (e.g., diabetes and hypertension) that are common in clinical settings

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EGCG-related differentially expressed genes were significantly enriched in the regulation of protein modifications, sterol biosynthesis, regulation of the MAPK cascade, neutrophil chemotaxis, granulocyte chemotaxis, neutrophil migration, and other functions (Figure 7F)