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In particular, our study showed that the intracellular depletion of GSH is able to induce cellular stress in NO-producing cells through a NO-dependent mechanism, such as inhibition of CcOX activity, DNA damage, and S-NO and 3-nitrotyrosine (NO 2 -Tyr) protein accumulation ( Figure 1 )

Interventions targeting ferroptosis attenuate neuroinflammation via reduced immune cell infiltration, while suppression of inflammation reciprocally diminishes neuronal ferroptosis

To further elucidate the phylogenetic mechanisms and conserved genomic structure of the AsGST gene, we examined the covariance between A

Patients were stratified based on the exposure variable of interest, which was the day of TIPS, classified as weekend vs