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For instance, TREM2 is a key receptor regulating microglial function and a marker protein of DAM phenotype, and its deficiency impairs the ability of microglia to respond effectively to myelin damage, thereby promoting demyelination and neurodegeneration (Poliani et al., 2015)

Non-oxidative loss of glutathione in apoptosis via GSH extrusion

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On the other hand, in MTC, biochemical recurrence does not always equate to the emergence of structural recurrence [164]