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Hepatocyte SH3RF2 deficiency is a key aggravator for nonalcoholic fatty liver disease

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Certainly, such coupling links cellular zinc to the cellular redox state, as a shift to more oxidizing conditions results in the release of zinc, whereas a shift to more reducing conditions results in its binding (192)

dSpCas9-VPR) targeted to the SNCA transcription start site (TSS) increases -syn mRNA and protein, whereas repression with dCas9-Krppel-associated box (KRAB) (dSpCas9-KRAB) in the same system decreases both transcripts and protein