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Likewise, SARS-Cov-2 infection and acute COVID-19 are accompanied by an inflammatory response [29], increased MPO activity [45], indicants of oxidative damage [46,47,48] and increased NO production including increased levels of nitrotyrosine [49], and lowered antioxidant defenses (ANTIOX) as indicated by reduced TAC levels [45, 49], Gpx [50], and zinc [51]

Because H 2 O 2 increases Nrf2 binding to DNA, a potential neuroprotective mechanism is that transient elevation of H 2 O 2 and altered redox states from the KD serve to activate Nrf2 leading to induction of antioxidation pathways (60)

Moura A, Tomada I, Tomada N