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Therefore, our results, in both humans and rats, relating individual differences in accumbal GSH content to the capacity to succeed in task performance over time, supports the view that higher individual GSH levels provide an advantage to deal with oxidative processes generated by the drastically enhanced energy demand - involving increased O 2 utilization-required in NuAc circuits to maintain performance (see above)

Fat mass and obesity-associated protein alleviates cerebral ischemia/reperfusion injury by inhibiting ferroptosis via miR-320-3p/SLC7A11 axis

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Differences between the metabolism of healthy cells and that of COPD cells are shown in Table 3