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The DSC results show that the thermal stability of the mutant is slightly higher than wild type, consistent with helix alpha2 forming new interactions with the other subunit

Although ATP13A2s function in mammalian cells remains elusive, loss-of-function mutations in ATP13A2 cause Kufor-Rakeb Syndrome (KRS), an autosomal recessive form of early-onset Parkinsonism with pyramidal degeneration and dementia (Ramirez et al., 2006)

It works by managing the activity of melanin-producing enzymes, helping the skin appear brighter and more uniform