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[DOI] [Google Scholar] 58.Ma X., Zhang Y., Li X., Bi J., Zhang G., Hao H., Hou H

This provides a new technical approach for establishing a mouse model of CYP2d6-induced Type 2 AIH

Macromolecular damage via oxidative and/or nitrosative stress, if unchecked, can theoretically contribute to disease development, and an increasing amount of evidence suggests that oxidative and/or nitrosative stress is linked to the pathophysiologic mechanisms of a myriad of human diseases [3,6,4850]