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8 , e2713e2713 (2017)

Loss of PTEN function further enhances glycolysis via AKT1 activation, stabilizing phosphofructokinase (PFKP)

Beyond editing, strategies are being developed to selectively remove large portions of mutated mtDNA by reconstituting double-strand break repair pathways within mitochondria, potentially eliminating deletion-bearing genomes (171)

Particularly, some sedative and analgesic anesthetics have been widely concerned as treatment methods to reduce oxidative stress and protect important organ functions