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HIF1 Represses Cell Stress Pathways to Allow Proliferation of Hypoxic Fetal Cardiomyocytes

shows that a combinatorial complex between estrogen receptor (ER)- and endothelial nitric oxide synthase (eNOS) could repress transcription of prognostic genes that are down-regulated in prostate tumors, such as the glutathione transferase gene GSTP1 (Re et al., 2011)