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The process of wound healing is intricate and cascaded, particularly in chronic wounds like severe burns and diabetic wounds, where unfavorable endogenous or external variables can impede the healing process by interfering with the regulation of inflammation [37]

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SIRT1 deacetylates Sterol Regulatory Element-Binding Protein-1c (SREBP-1c) and Carbohydrate Response Element-Binding Protein (ChREBP), suppressing the expression of their downstream lipogenic genes fatty acid synthase (FAS) and acetyl-CoA carboxylase 1 (ACC1), ultimately inhibiting lipogenesis and mitigating MAFLD

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