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Further, the crystal structure of NLRP3 revealed a disulfide bond between conserved cysteine residues in the inflammasome active site, suggesting redox-sensitive activity (Zhou et al., 2011

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Based on the analyzed data, we propose that the resulting phenotype produced by interfering with the gene of interest may not solely be due to the disruption of that particular gene but due to the combined interference of the gene of interest and the associated non-coding elements