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It is postulated herein that the detrimental activation of autoimmune T cells by glutamate in MS could lead to: (1) Cytotoxicity in the CNS: T cell-mediated killing of neurons and glia cells, which would subsequently increase the extracellular glutamate levels, and by doing so increase the excitotoxicity mediated by excess glutamate, (2) Release of proinflammatory cytokines, e.g., TNF and IFN that increase neuroinflammation

10.1007/s10068-012-0133-y 101 LeeJ.JangD

Contact your GP if you experience persistent fatigue, neurological symptoms (numbness, tingling, balance problems), memory difficulties, glossitis, or unexplained anaemia