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In accordance with this inhibition of vasorelaxation by reactive oxygen species, moderate hyperhomocysteinemia-induced endothelial dysfunction in mice heterozygous for a CBS defect was associated recently with an increased oxidant stress (Eberhardt et al, 2000)

While most EVs research has focused on mammalian cells, EVs are also secreted by bacteria and plant cells 1,2

Lahm A, Paolini C, Pallaoro M, Nardi MC, Jones P, Neddermann P, et al

The following antibodies were used: anti-GPX4(1:2000), anti-4 Hydroxynonenal(1:1000, ab46545, Abcam, USA), anti-ACSL4(1:2000), anti-actin(1:2000, 4967, Cell Signaling Technology, USA)